Keratoconus affects about 1 in 2,000 people. In some cases, when the cornea becomes dangerously thin or when sufficient vision can no longer be achieved by contact lenses due to steepening of the cornea, scarring or lens intolerance, corneal cross-linking is not an option and a corneal transplant may be required. In 2016, the FDA approved corneal collagen cross-linking to halt the progression of keratoconus. In most people the disease stabilizes after a few years without severe vision problems. As the disease progresses, special contact lenses (such as scleral contact lenses) may be required. Initially the condition can typically be corrected with glasses or soft contact lenses. Keratoconus causes very distinctive changes in the appearance of these maps, which allows doctors to make the diagnosis. Topography measures the curvature of the cornea and creates a colored "map" of the cornea. The underlying mechanism involves changes of the cornea to a cone shape. Proposed environmental factors include rubbing the eyes and allergies. Patients with a parent, sibling, or child who has keratoconus have 15 to 67 times higher risk in developing corneal ectasia compared to patients with no affected relatives. While the cause is unknown, it is believed to occur due to a combination of genetic, environmental, and hormonal factors. In more severe cases a scarring or a circle may be seen within the cornea. This may result in blurry vision, double vision, nearsightedness, irregular astigmatism, and light sensitivity leading to poor quality-of-life. Keratoconus ( KC) is a disorder of the eye that results in progressive thinning of the cornea. Blurry vision, nearsightedness, light sensitivity
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